The site of compression is by a muscle previously not known to cause radial nerve palsy. We believe that this is the first report of compressive neuropathy of the radial nerve in a bodybuilder.
6 Radial nerve entrapment after muscular overexertion 7, 8 has also been reported to be caused by a fibrous arch coming from the long head of the triceps. 5 In addition, progressive palsy has been reported as a result from compression of the triceps in the absence of any defined fibrous arch. 1– 4 The latter has been attributed to compression of the nerve as it passes through the distal portion of the spiral groove by a fibrous arch in the lateral head of the triceps. Radial nerve palsy may occur with humeral shaft fractures, particularly oblique fractures at the junction of the middle and distal thirds of the bone, local trauma, and after strenuous exercise. A review after six months showed that radial nerve function had been completely restored (fig 2). He was started immediately on a physiotherapy regimen to mobilise his right arm. This finding showed that the level of entrapment was at the triangular space, which compromised the nerve supply to the lateral head of the triceps, which has a lower origin. The long head and medial head of the triceps were spared because of the high origin of the branches of the radial nerve. The lateral head of the triceps was extremely irritable to the point of spontaneously twitching after the decompression was performed.
RADIA NERVE FREE
The aponeurotic edge of the muscle was incised 1.5 cm proximally allowing free passage of the examining digit into the triangular space until no extrinsic compression was felt. The nerve was dissected proximally to the inferior edge of the teres major, which appeared to be constricting the underlying nerve.
There was a notable extensive venous engorgement of the comitantes vessels. The course of the nerve was traced proximally. The raphe was incised in the usual manner to expose the nerve.
The radial nerve was identified after entering the interval between the long and lateral heads of the triceps. The patient was placed in the lateral decubitus position, and a posterior longitudinal incision (fig 1) was made. As no recovery was noted after four months, exploration of the right radial nerve was planned. Magnetic resonance imaging of the right arm did not show any abnormal soft tissue mass, with a normal appearance of the humeral cortex and marrow signal. He was reviewed again after three months no improvement was found. In view of spontaneous recovery of a previous compressive neuropathy without surgical intervention, a similar approach of conservative treatment was started with non-steroid anti-inflammatory drugs and physiotherapy. A nerve conduction study showed severe acute motor denervation of muscles innervated by the right radial nerve, suggesting a lesion around the spiral groove. Plain radiography did not show any sinister bony and soft tissue lesion. Physical examination showed considerable wasting of the brachioradialis muscle, loss of the extensor carpi radialis and extensor carpi ulnaris, loss of digital extensors, and loss of the extensor pollicis longus and abductor pollicis longus. Past medical history included left sided carpal tunnel syndrome, which resolved spontaneously in six months. There was no evidence of recent trauma to the brachial plexus, upper arm, or forearm to account for the condition. A 45 year old, right handed, elite, male bodybuilder presented with a two week history of gradual onset of spontaneous right wrist drop.
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